Stress causes
- Atrophy
- Hypertrophy
Stress
Physical forces applied to the body as well as psychological, social, and emotional factors
Physical stress theory
Body+Tissues react to the stress forces placed on them in a predictable manner
Stress causes: Atrophy
Relative level of stress falls below the maintenance level
Stress causes: hypertrophy
Duration and magnitude of the stress is progressively
increased
Mechanisms of Injury
- History portion of clinical examination (patient describes what happened)
- Macrotrauma
- microtrauma
MOI: Macrotrauma
Microtrauma
§ Occurs with repeated submaximal forces
over
time, and the tissues are unable to adapt
Occurs when a single force exceeds the
tissue’s failure point
MOI: Microtrauma
Occurs with repeated submaximal forces over time, and the tissues are unable to adapt
Forces: Tensile forces
- Longitudinal “tearing”
stress - Muscles,
tendons,
ligaments and fascia are
prone - Dynamic
overload
- Muscle is contracting
eccentrically while an
antagonistic force
attempts to elongate
the muscle.
- Muscle is contracting
Forces list
- tensile forces
- compression forces
- shear forces
- torsion forces
- direct blow
Soft tissue pathology: strutures
- Muscles and tendons
- Skin
- Joint capsules
- Ligaments
- Nerve
- Bursae
Soft tissue pathology: cause
- Loss of motion
- Decreased strength
- Decrease volitional control
- Create joint instability
Musculotendinous Injuries: Partial muscle or tendon tears
- Affects the muscle’s ability to generate or sustain tension
- Decreased force production
- Secondary to pain
Strains
- Noncontact injuries of the
musculotendinous unit - Caused by
- Tensile forces
- Dynamic overload
- Eccentric muscle contraction
- (Example: Lowering a weight).
Tendinitis
Inflammation of the
structures encased within
the tendon’s
outer layer
Tendinosis
- Degenerative condition
- Little evidence of healing
- Most common at the bony attachment
- More
prevalent than
tendinitis (but often occur
together)
Mechanisms Leading to Tendinopathy: Microtrauma
- Repetitive tensile loading, compression, and abrasion of the working tendons
- insufficient rest periods = accumulation of themicrotrauma ==> tendon failure.
Mechanisms Leading to Tendinopathy: Macrotrauma
- A single force placed on the muscle, discrete tearing within the tendon or at the musculotendinous junction.
- area
becomes the weak link
- forces of otherwise
normal
activity are sufficient to cause further inflammation.
- forces of otherwise
normal
Mechanisms Leading to Tendinopathy: Biomechanic Alteration
- redistribution of
the forces around a joint, => new tensile loads, compressive forces, or wearing of the tendons. - Ex. running on uneven terrain or using poor technique
Contusions
- Soft tissue injuries resulting from direct blows
- Capillary bleeding and breakdown of hemoglobin
- Dark purple and blue
- Superficial bone contusions are more painful
- Eliminate underlying injuries
- Fractures
- Superficial nerve damage
- Treatment
- Ice, gentle stretching, and padding
Bursae
fluid-filled sacs that buffer tissues from
friction-causing
structures to facilitate smooth motion
Bursitis
- inflammation of the bursae
- Caused by
- Disease state
- Increased stress
- Friction
- Traumatic force
- only palpable when inflamed
Heterotropic Ossification
- Formation of bone within a muscle belly’s fascia
- Occurs secondary to traumatic injury
- MRI—identifies intramuscular hematoma
- Aspirate to restore function
- Commonly occurs in
- Quadriceps femoris
- Hip adductor group
- Biceps brachii
- Can lead to compartment syndrome
- ROM is usually affected
Compartment Syndromes: Acute
- Result from trauma (contusions
and fractures - Considered medical
emergency(absent pulse= immediate danger)
Compartment Syndromes: Chronic
- exertional/recurrent
compartment syndromes - Occurs
during exercise;
muscles enlarge but
compartment cannot expand - Leg and forearm
compartments common
locations
Compartment Syndromes: Signs and Symptoms
- Pain, numbness, or paresthesia in the distal extremity
Compartment Syndromes: Five P's
- Pain
- Pallor (redness)
- Pulselessness
- Paresthesia
- Paralysis
Sprains
- Joint is forced beyond anatomical limits.
- Stretching or tearing of ligaments or capsule
- Ligaments in capsule = produce more swelling.
Degrees of Sprains
- First degree (little stretched, firm end-point, local pain+mild point)
- Second Degree (partial tearing, joint laxity, softish, mod. pain+swelling)
- Third degree (ruptured ligament, gross laxity, empty end point, complete loss of function)
Joint dislocations
- Disassociation of thejoint’s articulatingsurfaces
- Obvious deformity
- Open dislocation
- Joint surfaces protrudethrough the skin.
- Joint reduction should beperformed ASAP
Joint Subluxation
- Subtle chronic instability
- Joint surfaces that partially separate
- Joint that dislocates and
spontaneously
reduces
Synovitis
- Inflammation of a joint’s capsule
- Secondary to
existing inflammation thatspreads to the synovial membrane
- Synovial membrane—membrane lining a fluid-filledjoint
- Secondary to
existing inflammation thatspreads to the synovial membrane
- “Bogginess” within the joint
- Patient holds joint stressless position
Cellulitis
- Result of bacterial infection on the skin’s connective tissue
- Immediately refer to a physician
- Antibiotics
- Possible hospitalization
Cellulitis: Sign and Symptoms
- Edema
- Redness
- Tightening of the skin
- No outward leakage of pus
Articular Surface Pathology
- Hyaline cartilage
- Found on the articular surface of
bones (withstand compressive
+shearing forces) - Acutely injured or degenerative changes
- Irreversible, results in chronic joint pain
Articular Surface Pathology: Signs and Symptoms
- Chronic joint pain
- Dysfunction
Osteochondral Defects (OCD)
Result from trauma or softening of bone
Juvenile osteochondritis
- Affects patients younger than 15 years of age
- Adult—over 15 years of age
Osteochondritis dissecans
- Bony fragment—stable or free-floating
- Signs and
symptoms
- Pain
- Loss of ROM
- Decreased function
- “Joint locking”
Arthritis
- Degeneration of a joint’s articular surface
- Osteoarthritis is most common type found in
athletes - Affects weight-bearing joints
Rheumatoid arthritis
- Autoimmune condition that affects the articular
cartilage of multiple joints - Joint appears to be hard and nodular
Juvenile rheumatoid arthritis
- (6 months–16 years of
age) - Unexplained joint pain and swelling
- Joint rashes and high fever
Exostosis
- Growth of extraneous
bone at the site of
stress- Stress reaction
- Irregular forces
- Signs and symptoms
- Painful
- May limit movement
Apophysitis
- Inflammatory condition
involving a bone’s growth
plate- Tightness of muscles or
repetitive forces applied to
the bone - Inflammation/separation
of
muscle and bone
- Tightness of muscles or
Apophysitis: History of rapid growth spurt
- Muscular tissues fail
to adapt to the new bone
length - apply increased
stress to growth plate.
Nonunion fractures
Fracture that fails to heal within 9 months of expected time required
Malunion fractures
Healed fractures that are in a medically
unacceptable position
Avulsion Fractures
- Tearing away of a
ligament or tendon’s
bony attachment- Forceful muscle
contraction
- Forceful muscle
Avulsion Fractures: Signs and Symptoms
- Obvious deformity
- Tendon recoils
- Pain
- Tenderness
Peripheral Nerve Injury
- Entrapment injuries
- Common at ankle, elbow, wrist, and cervical spine
- Entrapped by tissue or swelling
Peripheral Nerve Injury: Signs and symptoms
- Dysfunction
- Paresthesia
- Muscular weakness
Neurovascular Pathologies
- Trauma to nerves, arteries, and veins
- Caused by
- Joint dislocations
- Bony displacement
- Concussive forces
- Compartment syndromes
- Can lead to loss of affected body part or loss of function
Stress Fractures: Fatigue fracture
- bone suspected to high, repeated submaximal stresses
- sudden changes to frequency, intensity, and duration
Stress Fractures: Insufficiency fracture
abnormally weak bones = subjected to normal forces