Assessment of the collapsed runner
ABCDEFGH
Airway
Breathing
Circulation
Disability (Mental Status)
Environment (Rectal Temperature)
Fluid status including change in body weight
Blood Glucose and Sodium
History including site of collapse
Collapse during exercise (Common medical causes)
• Cardiac arrest
• Hypoglycaemia
• Anaphylaxis
• Other medical conditions
• Trauma/Orthopaedic injuries
Collapse during exercise (exercise related causes)
• Heatstroke
• Hypothermia
• Exercise-associated hyponatraemia
• Exercise-associated postural hypotension
Most common cause of collapse Presents after the finish
Exercise-associated postural hypotension
Exercise-associated postural hypotension definition
“The inability to stand or walk unaided as the result of light headedness, faintness, dizziness or syncope”
- Postural hypotension related to cessation of activity
EAPH field management
- Place runner supine and head-down
- Oral fluids
Hyperthermia
Hyperthermia is high rectal temperature (usually >40oC) in an
athlete who, other than feeling exhausted, is usually well.
Exertional Heatstroke
- Symptoms or signs of organ dysfunction
- And a core body temperature >40oC
39-40oC is considered ...
is a normal physiological response to intense exercise or physical activity
Heat gain in exercise
Endogenous heat production: muscle activity and metabolism
+
Exogenous heat absorbtion: environmental
Heat loss in exercise
In low environmental temperatures, heat is lost through:
Convection + Radiation + (Small contribution from conduction)
Increasing environmental temperatures:
sweating = effective heat loss through evaporation
In Humid conditions:
evaporation is reduced > ineffective cooling
Athlete is unable to lose heat > begins to overheat
Heatstorke effects
Cerebral hyperthermia:
- hypothalamic failure and loss of thermoregulatory control
- Rhabdomyolysis
- renal failure
- hyperkalaemia
Intense inflammatory response:
Inflammatory cytokines trigger a systemic inflammatory response (SIRS):
- tissue hypoxia
- metabolic acidosis
- Acute kidney injury
- Acute liver failure
- Acute hypotensive shock
- Multi-system organ failure
third leading cause of death in athletes
- EHS
- Mortality and prognosis is largely dependent on early recognition and prompt management
Heatstroke: Field management
- Cold water immersion is the most effective cooling modality
- Shade, strip, spray and fan (S3F) is recommended where CWI is contraindicated or not possible
What is Exertional Heat Illness (EHI)
Pathophysiological continuum from mild symptoms (fatigue, headache) to collapse, coma and death because of a rise in core temperature
What is “Heat Exhaustion”
a physiological response to inadequate acclimatisation
What is “Heat cramps”
a physiological response to inadequate training
What is Exercise-associated hyponatraemia
[Na+] <135 mmol/L1
dilutional hyponatraemia occurring during or <24hr after exercise
EAH: pathophysiology
Relative hyponatraemia in the vascular compartment causes an osmotic fluid shift into adjacent tissues
EAH symptoms
Cerebral edema
- agitation
- LOC
- confusion
- Drowsiness
- stupor
GI symotoms
- nausea
- vomiting
- diarrhea
Other
- Headach
EAH causes
Excessive fluid intake
• Excessive consumption irrespective of fluid type
Reduced fluid output (Altered renal function)
- Exercise ( RBF and GFR fall by 40% after exercise)
- ADH (0.5 pg/mL) in 43 % of cases
- NSAIDs ( Indomethacin, and celecoxib significantly reduced free water clearance)
EAH: who is at risk?
- Females
- slower runers
- >4hrs exercise time
EAH: Field management (Asymptomatic)
● Observe closely
● Salty snacks or salty broth
● No oral fluids until onset of urination
● No iv fluids
EAH: Field management (Symptomatic)
● Oxygen
● 100ml bolus iv 3% Hypertonic Saline
● May require hospital admission
EAH: Hospital management (Symptomatic)
● Check and treat [Na+] before imaging
● Use 2.7% or 3% Hypertonic Saline
● AVOID Normal Saline
EAH: Prevention
1. Drink according to thirst: ad libitum
2. Avoid excessive drinking during and after exercise
3. Sports drinks do not prevent EAH
4. Sodium supplementation does not prevent EAH
Variation in human water turnover associated with environmental and lifestyle factors.
- age
- sex
- FFM
- Body weight
- PAL
- athletes
- HDI
- Air temprature
- Humidity
- Altitiude
EAH: drinking myths
1. We need to drink 2 liters of water a day
2. Sports drinks prevent EAH
3. Thirst is imperfect
Metabolic water
• The energy cost of running a marathon for an average
70 kg male is roughly 12,000 kJ (4.18 kJ·kg−1·min−1).
• Estimates of carbohydrate oxidation indicate that an
elite male runner would utilise 400 g of glycogen
• given the accepted value of 3 g of water per gram of
oxidised glycogen, this would result in a 1200 mL
endogenous water release