chapter 37- slides 60-119 (final gu exam)
Flank empaconis
flank bruising (tuner sign)
renal trauma diagnostic test
Polycystic kidney disease
hereditary
-multiple cysts in the kidney -progressive, no treatment
S/S- full flank pain or back pain, hematuria, hypertension, recurrent UTI
Nephrotic Syndrome
excretion of protein lost in urine from increased glomerular membrane permeability.
-liver produces lipoproteins; foamy urine
-decrease In albumin, decrease oncotic pressure fluid shifts from blood vessels into tissues
nephrotic causes
periorbital edema, dependent edema, ASCITES-why it matters? fluid overload, skin breakdown, and impaired mobility
hyperlipidedema
a common condition characterized by excessively high levels of of lipids- cholesterol triglycerides- In the blood often leading to dangerous plaque build up In the arteries
Nephrosclerosis
hypertension damgages kidneys by sclerotic changes. (reduces perfusion& damaging glomeruli)
glomerulonephritis
inflammatory disease of the glomerulus.
-proteins, WBC, RBC, leak into urine
Causes acute post-streptococcal infection
key findings
cola-colored urine (hematuria), periorbital/ generalized edema hypertension, decreased urine output, most common in children
actue kidney injury
sudden loss of kidney function
azotemia
waste products accumulate
kidneys cannot excrete nitrogenous waste- BUN and creatine rise
prorenal
not enough blood to the kindeys (ex: dehydration or shock)
intrarenal
kidney tissue is damaged (ex: nephrotoxic drugs)
post renal
urine can't get out due to blockage (ex:BPH)
What phase is the patient in when they put out massive amounts of urine?
diuretic phase
-risk for dehydration, hypokalemia and hypotension
Potassium is the most important in the kidneys, without correcting potassium, can cause cardiac dysthymia's
T/F
TRUE
Chronic kidney disease problems
uremia
the kidneys shut down (urea in the blood)
-affects all body systems
S/S for CKD
decreased calcium increased phosphors
When kidneys are failing, they are not producing erythropoietin
True
Therapeutic interventions for chronic kidney disease
why might we start dialysis ?
when the kidneys can no longer remove fluid, potassium, or toxins
hemodialysis
Artificial kidney removes waste products and excess waste from blood.
(hemodialysis)
ARTERIVENOUS (AV) FISTULA
lowest risk of infection
purposes of hemodialysis
Hyperkalemia with EKG changes is immediate lethal risk
(t/f)
True
A-E-I-O-U
what's true about hypotension?
it is a big issue after dialysis
Permanent vascular access care
If a patient comes back from dialysis and he's scheduled for a invasive procedure, what complications can happen?
can hemorrhage, NEED TO CALL HP to inform them on possible complications!
Peritoneal Dialysis
Peritoneal dialysis
patient can get peritonitis- use sterile technique while performing.
If fluid come back cloudy and abdominal pain is present
Think peritonitis
Kidney translant
will need to take anti rejection meds for the rest of your life.
for kidney transplant, they NEVER remove the old kidney unless
there is a sign of infection or cancer
-as soon as kidney is connected, there should be urine return .
what is the most common cause of CKD and end stage kidney disease?
diabetes
nephrotic syndrome
the excretion of 3.5 grams or more of protein in the urine per day .
Glomerulonephritis causes
can be caused by a variety of factors, including immunological abnormalities, infectious agents, systemic disease, toxins and vascular disorders
initiating phase of AKI
begins at the renal injury and lasts until the occurrence of symptoms
Oliguric phase oF AKI
less than 400 ml of urine is produced in 24 hours.
diuretic phase of AKI
the kidneys begin to excrete waste products again, 1 to 3 L/day of urine is produced.
Recovery phase AKI
in this final phase recovery begins as the GFR rises.