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Summer Immuno Lecture 13

front 1

A child has severe recurrent viral and fungal infections because thymic development failed. Which diagnosis best fits?

A. Wiskott-Aldrich syndrome

B. Hyper-IgM syndrome

C. DiGeorge syndrome

D. Canale-Smith syndrome

back 1

C. DiGeorge syndrome

front 2

In DiGeorge syndrome, absent thymic tissue most directly impairs development of which organ?

A. Thymus

B. Spleen

C. Bone marrow

D. Lymph node

back 2

A. Thymus

front 3

A patient with DiGeorge syndrome has profound cellular immune dysfunction. Which cell type is primarily affected?

A. B cells

B. Neutrophils

C. Macrophages

D. T cells

back 3

D. T cells

front 4

The life-threatening infections in DiGeorge syndrome are mainly due to loss of which immune function?

A. Antibody secretion

B. T-cell function

C. Complement activation

D. Neutrophil migration

back 4

B. T-cell function

front 5

Which sequence correctly lists the major phases of HIV infection?

A. Acute, latent, recovered

B. Acute, chronic, profound immunosuppression

C. Chronic, acute, latent

D. Prodrome, remission, recovery

back 5

B. Acute, chronic, profound immunosuppression

front 6

After HIV enters a target cell, which enzyme copies viral RNA into DNA?

A. DNA polymerase

B. RNA primase

C. Reverse transcriptase

D. RAG recombinase

back 6

C. Reverse transcriptase

front 7

Reverse transcriptase converts HIV genetic material into which product?

A. cDNA

B. mRNA

C. rRNA

D. tRNA

back 7

A. cDNA

front 8

After reverse transcription, which event allows HIV DNA to persist inside the host genome?

A. Capsid degradation

B. Ribosomal binding

C. Antibody neutralization

D. Insertion into host DNA

back 8

D. Insertion into host DNA

front 9

Once HIV DNA is inserted into host DNA and remains inactive, which effector cells fail to detect it?

A. B cells

B. NK cells

C. CTLs

D. Neutrophils

back 9

C. CTLs

front 10

A patient is born with nonfunctional CD40L. Which co-stimulatory pathway is directly defective?

A. CD28-B7

B. CD40-CD40L

C. Fas-FasL

D. IgE-Fc receptor

back 10

B. CD40-CD40L

front 11

Loss of functional CD40 or CD40L most directly prevents which immune response?

A. T-dependent antibody response

B. NK-cell cytotoxicity

C. Neutrophil oxidative burst

D. Complement membrane attack

back 11

A. T-dependent antibody response

front 12

A patient with defective CD40 signaling has impaired germinal-center function. Which two processes are compromised?

back 12

Class switching and hypermutation

front 13

Without functional CD40/CD40L signaling, antibody production is biased toward which class?

back 13

IgM

front 14

Which immunodeficiency syndrome eliminates both major adaptive lymphocyte arms?

back 14

SCID

front 15

Severe combined immunodeficiency involves dysfunction of which lymphocyte groups?

back 15

B and T cells

front 16

A patient with a congenital C3 defect has lymph nodes lacking which structure?

____ ____

back 16

Germinal centers

front 17

A C3-deficient patient lacks germinal centers. Which antibody class would B cells mainly produce?

back 17

A. IgM

front 18

Initial HIV transmission commonly begins after viral penetration through which barrier?

A. Respiratory epithelium

B. Keratinized skin

C. Gastric mucosa

D. Rectal or vaginal mucosa

back 18

D. Rectal or vaginal mucosa

front 19

After crossing mucosal surfaces, HIV initially infects which target cell population?

A. B cells

B. Neutrophils

C. T cells

D. Eosinophils

back 19

C. T cells

front 20

Once inside T cells, HIV replicates by taking over which resource?

A. Complement proteins

B. Host machinery

C. Granule enzymes

D. Antibody genes

back 20

B. Host machinery

front 21

During acute HIV infection, viral replication across the body does what?

A. Stops immediately

B. Remains localized

C. Clears completely

D. Multiplies systemically

back 21

D. Multiplies systemically

front 22

During acute HIV infection, viral levels typically peak at approximately what time?

A. 3-4 weeks

B. 2-3 days

C. 6-8 months

D. 1-2 years

back 22

A. 3-4 weeks

front 23

During the chronic phase of HIV infection, what happens to viral levels?

A. Disappear completely

B. Peak immediately

C. Decline but persist

D. Remain undetectable

back 23

C. Decline but persist

front 24

As HIV progresses toward profound immunosuppression, what ultimately explains susceptibility to fatal infections?

Loss of ____ cells

back 24

Loss of T cells

front 25

HIV-1 stores its genetic information in which nucleic acid form?

back 25

RNA

front 26

Why can killer T cells fail to detect HIV-infected cells during latency?

A. HIV destroys antibodies

B. HIV blocks all cytokines

C. HIV exits immediately

D. HIV remains hidden intracellularly

back 26

D. HIV remains hidden intracellularly

front 27

A newly infected patient asks whether HIV is detected immediately. Which feature explains delayed immune recognition?

A. Acute sterilization

B. Latent cellular buildup

C. Permanent antibody absence

D. Immediate viral clearance

back 27

B. Latent cellular buildup

front 28

During the latent period of HIV, infected cells mainly serve as what?

A. Complement reservoirs

B. Reactivatable viral reservoir

C. Neutrophil traps

D. IgE-producing cells

back 28

B. Reactivatable viral reservoir

front 29

Each HIV replication cycle introduces mutations. What is the immune consequence?

A. Easier T-cell recognition

B. Faster complement fixation

C. Reduced viral diversity

D. Harder immune recognition

back 29

D. Harder immune recognition

front 30

High mutation rates help HIV maintain which advantage?

A. Immune escape

B. Antibody deletion

C. Eosinophil recruitment

D. Mast-cell activation

back 30

A. Immune escape

front 31

Which set contains the major HIV-1 target cells?

A. B cells, eosinophils, neutrophils

B. NK cells, basophils, mast cells

C. Helper T cells, macrophages, dendritic cells

D. Platelets, fibroblasts, keratinocytes

back 31

C. Helper T cells, macrophages, dendritic cells

front 32

HIV infection of macrophages and dendritic cells is especially harmful because these cells normally perform which role?

A. Kill helminths directly

B. Produce thyroid hormone

C. Form epithelial barriers

D. Activate killer T cells

back 32

D. Activate killer T cells

front 33

One reason AIDS is deadly is that HIV can persist in which state?

A. Latent phase

B. Spore phase

C. Germinal phase

D. Vegetative phase

back 33

A. Latent phase

front 34

HIV spreads efficiently because it hijacks which system?

A. Endocrine system

B. Immune system

C. Skeletal system

D. Digestive system

back 34

B. Immune system

front 35

Untreated HIV-1 infection commonly leads to death within approximately how long?

A. 6 months

B. 2 years

C. 50 years

D. 10 years

back 35

D. 10 years

front 36

People with AIDS are treated with which medication class?

A. Antihistamines

B. Antiretrovirals

C. Glucocorticoids

D. Antifungals

back 36

B. Antiretrovirals

front 37

Some patients naturally control HIV-1 without typical progression. What are they called?

A. Elite controllers

B. Latent carriers

C. Viral amplifiers

D. Seronegative hosts

back 37

A. Elite controllers

front 38

In elite controllers, pattern-recognition receptor signaling increases secretion of which two antiviral cytokines?

back 38

IFN-alpha and IFN-beta

front 39

IFN-alpha and IFN-beta help elite controllers mainly by blocking which viral process?

A. Viral entry

B. Antibody production

C. Antigen presentation

D. Viral replication

back 39

D. Viral replication

front 40

Some elite controllers have stronger MHC I molecules. Which immune response improves?

A. Faster B-cell switching

B. Stronger IgE binding

C. Faster killer T activation

D. Increased eosinophil survival

back 40

C. Faster killer T activation

front 41

Better MHC I presentation in HIV elite controllers most directly activates which cell type?

back 41

Killer T cells

front 42

Which feature best explains why HIV is difficult to eliminate after initial infection?

A. Fixed surface antigens

B. No cellular reservoir

C. Immediate immune clearance

D. Latent reactivatable reservoir

back 42

D. Latent reactivatable reservoir

front 43

A virus infects APCs needed for cytotoxic T-cell activation. Which HIV target cell group explains this? A. Eosinophils and basophils B. Macrophages and dendritic cells C. Plasma cells and B cells D. Keratinocytes and fibroblasts

back 43

B. Macrophages and dendritic cells

front 44

A patient’s HIV mutates repeatedly as it replicates. Which outcome best follows?

A. Antigenic variation

B. Thymic regeneration

C. IgE blockade

D. Fas correction

back 44

A. Antigenic variation

front 45

Which combined features make AIDS especially deadly?

A. Low mutation, rapid clearance

B. IgE binding, mast activation

C. Latency, mutation, APC infection

D. Spore formation, toxin release

back 45

C. Latency, mutation, APC infection

front 46

Immune weakness can result from mutation of how many genes?

back 46

One gene

front 47

Compared with genetic immunodeficiencies, which category affects millions of people?

_______ immunodeficiencies

back 47

Acquired immunodeficiencies

front 48

Genetic immunodeficiencies are best described as which pattern?

A. Rare disorders

B. Universal disorders

C. Acquired disorders

D. Infectious disorders

back 48

A. Rare disorders

front 49

Which virus is most intensely studied as the cause of AIDS?

back 49

HIV-1

front 50

The abbreviation HIV-1 refers to which virus?

A. Human T-cell virus one

B. Human immunodeficiency virus one

C. Herpes immunodeficiency virus one

D. Host integration virus one

back 50

B. Human immunodeficiency virus one

front 51

During acute HIV infection, the dramatic rise in total circulating virus is called what?

Viral _____

back 51

Viral load

front 52

After acute HIV peaks, viral load decreases mainly because which cells begin working?

A. Virus-specific CTLs

B. Memory B cells

C. Follicular dendritic cells

D. Tissue mast cells

back 52

A. Virus-specific CTLs

front 53

Many viral infections fully clear after the acute phase. What is this clearance called?

_____

back 53

Sterilization

front 54

Unlike many viruses, full HIV-1 infection enters which long-term phase after acute infection?

A. Chronic phase

B. Germinal phase

C. Vegetative phase

D. Allergic phase

back 54

A. Chronic phase

front 55

During chronic HIV infection, which immune populations initially remain high?

_____ and _____ cells

back 55

CTLs and Th cells

front 56

During chronic HIV infection, Th-cell numbers slowly decrease because HIV does what?

_____ them

back 56

Kills them

front 57

In chronic HIV, CTLs eventually decline because they lose help from which cells?

_____ cells

back 57

Th cells

front 58

Late in untreated HIV, CTL decline causes which virologic change?

Viral ____ rises

back 58

Viral load rises

front 59

Profound immunosuppression in AIDS most directly predisposes to which fatal complication?

A. Seasonal allergy

B. Autoimmune arthritis

C. Opportunistic infection

D. Contact dermatitis

back 59

C. Opportunistic infection

front 60

HIV can initiate latent infection within approximately what time after exposure?

A. 24 hours

B. 3-4 weeks

C. 6 months

D. 5-10 days

back 60

D. 5-10 days

front 61

Latent HIV reservoirs may form before full activation of which system?

A. Innate immune system

B. Adaptive immune system

C. Complement system

D. Coagulation system

back 61

B. Adaptive immune system

front 62

Why does HIV latency form especially early relative to adaptive immunity?

A. Antibodies appear immediately

B. CTLs mature prenatally

C. Adaptation takes about one week

D. Neutrophils suppress latency

back 62

C. Adaptation takes about one week

front 63

HIV reverse transcriptase is especially dangerous because it is highly what?

____-prone

back 63

Error-prone

front 64

The error-prone nature of HIV reverse transcriptase produces which viral consequence?

High _____ rate

back 64

D. High mutation rate

front 65

HIV escape mutants can arise after approximately how long?

A. 3-4 weeks

B. 10 days

C. 2 years

D. 10 years

back 65

B. 10 days

front 66

Once HIV escape mutants arise, original CTLs usually fail to do what?

A. Produce antibody

B. Enter lymph nodes

C. Recognize them

D. Bind complement

back 66

C. Recognize them

front 67

Which molecule functions as HIV-1’s docking protein? _____

back 67

CD4

front 68

By infecting macrophages and dendritic cells, HIV damages cells needed for which process?

A. IgE crosslinking

B. CTL activation

C. Keratinocyte growth

D. Fas signaling

back 68

B. CTL activation

front 69

HIV-infected immune cells can carry virus from tissues to which site?

A. Bone marrow

B. Thymic cortex

C. Splenic red pulp

D. Lymph nodes

back 69

D. Lymph nodes

front 70

Within lymph nodes, HIV can undergo which change?

Faster ________

back 70

Faster proliferation

front 71

In lymph nodes, HIV may be coated by antibodies or which other system?

A. Coagulation

B. Kallikrein

C. Complement

D. Fibrinolysis

back 71

C. Complement

front 72

Opsonized HIV in lymph nodes can become trapped on which stromal immune cell?

A. Macrophages

B. Follicular dendritic cells

C. Mast cells

D. Neutrophils

back 72

B. Follicular dendritic cells

front 73

ART improves survival but does not do what?

A. Eliminate HIV

B. Block replication

C. Target viral enzymes

D. Slow progression

back 73

A. Eliminate HIV

front 74

ART works mainly by targeting components of which process?

A. Antibody class switching

B. Complement activation

C. Viral replication cycle

D. T-cell thymic selection

back 74

C. Viral replication cycle

front 75

The main clinical benefit of ART is best described how?

A. Sterilizes latent reservoirs

B. Lengthens patient survival

C. Restores thymus completely

D. Prevents all cancers

back 75

B. Lengthens patient survival

front 76

Patients on ART have increased risk of cancer and which neurologic complication?

_____ disorders

back 76

Cognitive disorders

front 77

_____-term ART-treated HIV patients have increased risk affecting kidneys, liver, bone, and the heart.

back 77

long

front 78

Average lifespan on ART is approximately how much shorter than uninfected individuals?

A. 5 years

B. 10 years

C. 15 years

D. 20 years

back 78

D. 20 years

front 79

Approximately what fraction of HIV-infected individuals are elite controllers?

A. 3%

B. 0.3%

C. 10%

D. 30%

back 79

B. 0.3%

front 80

Some elite controllers can control HIV infection for as long as what duration?

A. 30 years

B. 10 days

C. 3-4 weeks

D. 5-10 days

back 80

A. 30 years

front 81

In some elite controllers, CTLs are described as unusually what?

A. Suppressed

B. Vicious

C. Latent

D. Opsonized

back 81

B. Vicious