front 1 WHAT ARE THE 3 STAGES OF SHOCK | back 1
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front 2
| back 2 COMPENSATORY |
front 3 REVIEW SLIDE 12 ! NURSING MANAGEMENT IN EARLY SHOCK | back 3 REVIEW! |
front 4 EARLY SIGNS OF SHOCK! | back 4
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front 5
| back 5 PROGRESSIVE STAGE |
front 6 MANAGEMENT OF PROGRESSIVE STAGE: | back 6
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front 7
S/S
| back 7 IRREVERSIBLE STAGE |
front 8 NURSING MANAGEMENT IN IRREVERSIBLE SHOCK | back 8
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front 9 CRITICAL CONDITION WHERE THE BODY HAS DECREASED TISSUE PERFUSION EVENTUALLY LEADING TO ORGAN FAILURE AND DEATH | back 9 SHOCK |
front 10 CAUSED BY WIDE SPREAD BLOODBORNE INFECTION
| back 10 SEPTIC SHOCK |
front 11 SEPTIC SHOCK MANAGEMENT | back 11 INTERVENTIONS
FLUD REPLACEMENT PHARMACOLOGIC
NUTRITION
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front 12 CAUSED BY SPINAL CORD INJURY THAT IS T-6 OR HIGHER | back 12 NEUROGENIC SHOCK |
front 13 MAIN DIFFERENCE IN NEUROGENIC SHOCK VS ANY OTHER SHOCK! | back 13
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front 14 NEUROGENIC SHOCK MANAGEMENT | back 14
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front 15 CAUSED BY BLOOD LOSS FROM TRAUMA OR A GUNSHOT WOUND OR EVEN SURGERY OR BURNS
LOW BLOOD VOLUME | back 15 HYPOVOLEMIC SHOCK |
front 16 MANAGMENT OF HYPOVOLEMIC SHOCK | back 16
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front 17 THE HEART FAILS TO PUMP, SIMILAR TO HEART FAILURE OR MI, WHERE HEART MUSCLES ARE WEAK AND FAIL TO PUMP
| back 17 CARDIOGENIC SHOCK |
front 18 MANAGMENT OF CARDIOGENIC SHOCK | back 18
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front 19 WHAT ARE SOME LABS YOU WANT TO CHECK FOR PATIENTS W/ CARDIOGENIC SHOCK? | back 19
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front 20 SEVERE ALLERGIC REACTION
S/S
| back 20 ANAPHYLACTIC SHOCK |
front 21 MANAGEMENT OF ANAPHYLACTIC SHOCK | back 21
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front 22 CAUSED BY WIDESPREAD INFECTION MUST identify and tx source of infection SEVERELY LOW BP RESULTING IN ORGAN FAILURE TO LACK OF PERFUSION COOL CLAMMY SKIN DELAYED CAP REFILL MENTAL STATUS CHANGE - CONFUSION AND DISORIENTATION DUE TO LOW O2 PERFUSION HIGH WBC TEMP - VERY HIGH OR VERY LOW (<96) | back 22 SEPTIC SHOCK |
front 23 WHAT DIFFERENTIATES NEUROGENIC SHOCK FROM THE REST OF THE DIFFERENT TYPES OF SHOCKS | back 23
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front 24 INTERVENTIONS FOR NEUROGENIC SHOCK | back 24
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front 25 MOST IMPORTANT COMPLICATION OF NEUROGENIC SHOCK | back 25 AUTONOMIC DYSREFLEXIA CAUSED BY ANYTHING W/ THAT CAUSES CONSTRICTION:
S/S: HYPERTENSION |
front 26 PRIORITY S/S FOR HYPOVOLEMIC SHOCK | back 26 COLD/CLAMMY SKIN |
front 27 WHAT IS THE MOST COMMONLY USED ISOTONIC IV FLUID USED FOR SHOCK? | back 27 LR IS MOST COMMONLY USED; NS IS USED AS WELL LR ACTS AS A VOLUME EXPANDER FOR BETTER PERFUSION |
front 28 WHAT MUST A PATIENT HAVE ON FILE WHEN THEY HAVE SHOCK? | back 28 ADVANCED DIRECTIVES |
front 29 AVERAGE PRESSURE AT WHICH BLOOD MOVES THROUGH THE VASCULATURE | back 29 MEAN ARTERIAL PRESSURE |
front 30 WHAT IS A NORMAL MAP? | back 30 65 OR BETTER |
front 31 RESULT OF INADEQUATE PERFSION IS ANAEROBIC METABOLISM AND BUILD UP OF THIS?
| back 31 LACTIC ACID |
front 32 WHAT IS THE CRITERIA FOR LACTIC ACID TO BE FOR A PERSON TO BE CONSIDERED "IN SHOCK?" | back 32 GREATER THAN 2 |
front 33 WHAT IS THE INITIAL NURSING ACTION BEFORE ADMINISTERING BROAD SPECTRUM ABX? | back 33 GET A CULTURE |
front 34 REVIEW SLIDE 22 | back 34 REVIEW |
front 35 IMPROVE CONTRACTILITY OF HEART, INCREASED STROKE VOLUME, AND INCREASE CARDIAC OUTPUT
| back 35 INOTROPIC AGENTS
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front 36 REDUCE PRELOAD AND AFTERLOAD OF CARDIAC SYSTEM AND REDUCE O2 DEMAND OF HEART
| back 36 VASODILATORS
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front 37 INCREASE BP BY VASOCONSTRICTION
| back 37 VASOPRESSOR AGENTS
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front 38 RULES FOR VASOPRESSORS | back 38
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front 39 WHAT IS THE DAILY CALORIES NEEDED FOR PATIENTS W/ SHOCK? | back 39 AT LEAST 3000 CALOIRIES DAILY DU ETO INCREASED METABOLIC RATES
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front 40 STRESS ULCER PREVENTION DURING PROGRESSIVE STAGE | back 40 H2 BLOCKERS, ANTACIDS, PPIs |
front 41 VASOPRESSORS ARE USED TO KEEP THE MAP ABOVE WHAT? | back 41 65 |