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Phys 77

front 1

The two major hormones secreted by the thyroid gland are:
A. Thyroxine and triiodothyronine
B. Cortisol and aldosterone
C. Insulin and glucagon
D. Calcitonin and PTH

back 1

A. Thyroxine and triiodothyronine

front 2

Complete lack of thyroid secretion usually causes basal metabolic rate to fall:
A. 10–20% below normal
B. 60–70% below normal
C. 40–50% below normal
D. 5–10% below normal

back 2

C. 40–50% below normal

front 3

Thyroid secretion is controlled primarily by:
A. ACTH from anterior pituitary
B. TSH from anterior pituitary
C. TSH from posterior pituitary
D. TRH from thyroid gland

back 3

B. TSH from anterior pituitary

front 4

In addition to T3 and T4, the thyroid gland also secretes:
A. Somatostatin
B. Aldosterone
C. Erythropoietin
D. Calcitonin

back 4

D. Calcitonin

front 5

Of the metabolically active hormones secreted by the thyroid gland, about 93% is:
A. Thyroxine
B. Triiodothyronine
C. Calcitonin
D. Thyroglobulin

back 5

A. Thyroxine

front 6

Triiodothyronine (T3) is approximately how much more potent than thyroxine (T4)?
A. Ten times more potent
B. Two times more potent
C. Four times more potent
D. Half as potent

back 6

C. Four times more potent

front 7

The thyroid gland is composed of large numbers of closed structures called:
A. Acini
B. Follicles
C. Islets
D. Lobules

back 7

B. Follicles

front 8

The major constituent of thyroid colloid is:
A. Albumin
B. Transthyretin
C. Thyroperoxidase
D. Thyroglobulin

back 8

D. Thyroglobulin

front 9

Biopsy highlights parafollicular “C cells.” Their key secretion is:
A. Thyroxine
B. Triiodothyronine
C. Calcitonin
D. Thyroglobulin

back 9

C. Calcitonin

front 10

After oral ingestion, iodides are primarily:
A. Secreted into bile
B. Converted to iodate
C. Excreted in stool
D. Absorbed into blood

back 10

D. Absorbed into blood

front 11

The first stage in thyroid hormone formation is:
A. Iodination of thyroglobulin
B. Transport iodide into thyroid
C. Proteolysis of thyroglobulin
D. Deiodination of T4

back 11

B. Transport iodide into thyroid

front 12

The sodium-iodide symporter transports:
A. One sodium with one iodide
B. Two iodide with one sodium
C. Two sodium with one iodide
D. One chloride with one iodide

back 12

C. Two sodium with one iodide

front 13

The process of concentrating iodide inside thyroid cells is called:
A. Iodide trapping
B. Iodide organification
C. Iodide coupling
D. Iodide recycling

back 13

A. Iodide trapping

front 14

Iodide is transported across the apical membrane into the follicle by:
A. Basolateral iodide efflux pump
B. Apical sodium-iodide symporter
C. Apical iodide-protein transporter
D. Apical chloride-iodide exchanger

back 14

D. Apical chloride-iodide exchanger

front 15

The thyroid’s hormone-secreting epithelial cells are best described as:
A. Typical protein-secreting gland cells
B. Typical steroid-secreting gland cells
C. Typical neurotransmitter-secreting cells
D. Typical exocrine ductal cells

back 15

A. Typical protein-secreting gland cells

front 16

Each molecule of thyroglobulin contains about:
A. About 10 tyrosines
B. About 70 tyrosines
C. About 700 tyrosines
D. About 25 tyrosines

back 16

B. About 70 tyrosines

front 17

The first essential chemical step enabling hormone formation is to:
A. Oxidize iodide for tyrosine binding
B. Store T3 in vesicles
C. Convert T4 into T3
D. Export T4 via pendrin

back 17

A. Oxidize iodide for tyrosine binding

front 18

Oxidation of iodine is promoted by:
A. Deiodinase with NADPH
B. Thyroglobulin with iodine
C. Peroxidase with hydrogen peroxide
D. Pendrin with chloride gradient

back 18

C. Peroxidase with hydrogen peroxide

front 19

Thyroid follicles are lined primarily by:
A. Squamous epithelial cells
B. Cuboidal epithelial cells
C. Columnar epithelial cells
D. Transitional epithelial cells

back 19

B. Cuboidal epithelial cells

front 20

Thyroglobulin contains about 70 of which amino acid residues?
A. Lysine residues
B. Cysteine residues
C. Tryptophan residues
D. Tyrosine residues

back 20

D. Tyrosine residues

front 21

Iodide exits thyroid cells into the follicle lumen via:
A. Sodium-iodide symporter
B. Pendrin
C. Peroxidase
D. Thyroglobulin

back 21

B. Pendrin

front 22

A newborn has a hereditary absence of the thyroid peroxidase system. Thyroid hormone synthesis rate falls to:
A. Decreases modestly
B. Falls to one-third
C. Falls to half
D. Falls to zero

back 22

D. Falls to zero

front 23

The binding of iodine to the thyroglobulin molecule is termed:
A. Organification of thyroglobulin
B. Iodide trapping
C. Coupling reaction
D. Deiodination

back 23

A. Organification of thyroglobulin

front 24

During iodination, tyrosine is converted first to:
A. DIT then MIT
B. T4 then T3
C. MIT then DIT
D. T3 then T4

back 24

C. MIT then DIT

front 25

Two molecules of diiodotyrosine couple to form:
A. Thyroxine (T4)
B. Triiodothyronine (T3)
C. Reverse T3
D. Thyroglobulin

back 25

A. Thyroxine (T4)

front 26

One MIT plus one DIT couples to form:
A. Thyroxine (T4)
B. Reverse T3
C. Diiodotyrosine
D. Triiodothyronine (T3)

back 26

D. Triiodothyronine (T3)

front 27

The thyroid gland is unusual among endocrine glands because it:
A. Has minimal storage capacity
B. Stores large hormone amounts
C. Stores only calcitonin
D. Stores only iodide

back 27

B. Stores large hormone amounts

front 28

Most thyroglobulin is ______ into circulating blood:
A. Rapidly released
B. Slowly released
C. Not released
D. Pulsatile release

back 28

C. Not released

front 29

During hormone release, apical pseudopods primarily form:
A. Pinocytic vesicles
B. Secretory granules
C. Tight-junction pores
D. Ribosomal channels

back 29

A. Pinocytic vesicles

front 30

Lysosomes fuse with pinocytic vesicles to form:
A. Storage vesicles
B. Peroxisomes
C. Digestive vesicles
D. Golgi cisternae

back 30

C. Digestive vesicles

front 31

The key enzymes freeing T3/T4 from thyroglobulin are:
A. Lipases
B. Kinases
C. Polymerases
D. Proteases

back 31

D. Proteases

front 32

Some colloid thyroglobulin enters thyroid cells via:
A. Diffusion through colloid
B. Megalin-mediated endocytosis
C. Pendrin cotransport
D. NIS symport

back 32

B. Megalin-mediated endocytosis

front 33

About what fraction of iodinated tyrosines remain MIT/DIT, never becoming thyroid hormones?
A. One tenth
B. One third
C. Three quarters
D. All of them

back 33

C. Three quarters

front 34

Deiodinase enzymes primarily:
A. Couple MIT with DIT
B. Trap iodide in cell
C. Oxidize iodide
D. Recycle iodine within gland

back 34

D. Recycle iodine within gland

front 35

Lack of deiodinase enzymes can cause:
A. Iodine deficiency
B. Hypercalcemia
C. Iron deficiency
D. Zinc deficiency

back 35

A. Iodine deficiency

front 36

On entering blood, T3/T4 bind immediately to:
A. RBC hemoglobin
B. Plasma proteins
C. Platelets
D. Immunoglobulins

back 36

B. Plasma proteins

front 37

The major thyroid hormone–binding proteins are:
A. CBG, transferrin, albumin
B. SHBG, albumin, haptoglobin
C. TBG, albumin, haptoglobin
D. TBG, transthyretin, albumin

back 37

D. TBG, transthyretin, albumin

front 38

After a large thyroxine injection, metabolic effects are minimal for:
A. Two to three days
B. Two to three hours
C. Ten to twelve days
D. Immediate effect

back 38

A. Two to three days

front 39

Thyroxine reaches maximal effect at about:
A. 24 hours
B. Ten to twelve days
C. Three to four days
D. Several months

back 39

B. Ten to twelve days

front 40

The general effect of thyroid hormone is to:
A. Block ribosomal translation
B. Open calcium channels
C. Activate nuclear gene transcription
D. Inhibit cAMP formation

back 40

C. Activate nuclear gene transcription

front 41

Most thyroxine secreted is converted to:
A. Reverse T3
B. Triiodothyronine (T3)
C. Calcitonin
D. Thyroglobulin

back 41

B. Triiodothyronine (T3)

front 42

About 90% of receptor-bound thyroid hormone is:
A. Thyroxine
B. Reverse T3
C. Diiodotyrosine
D. Triiodothyronine

back 42

D. Triiodothyronine

front 43

Thyroid hormone receptor usually heterodimerizes with:
A. Retinoid X receptor
B. Vitamin D receptor
C. Estrogen receptor
D. Glucocorticoid receptor

back 43

A. Retinoid X receptor

front 44

TR/RXR binds DNA at:
A. CpG islands
B. TATA boxes
C. Thyroid response elements
D. Splice donor sites

back 44

C. Thyroid response elements

front 45

A researcher infuses TSH into isolated thyroid follicles. Which immediate change most directly increases hormone release?
A. Increased thyroglobulin exocytosis
B. Decreased lysosomal enzyme activity
C. Increased proteolysis of thyroglobulin
D. Decreased colloid endocytosis

back 45

C. Increased proteolysis of thyroglobulin

front 46

Patient with low thyroid hormone has elevated TSH. Which transport step is most upregulated in thyroid cells?
A. Increased iodide pump activity
B. Decreased apical iodide efflux
C. Reduced thyroid peroxidase activity
D. Increased hepatic T4 clearance

back 46

A. Increased iodide pump activity

front 47

In a euthyroid patient given exogenous TSH, which synthetic step increases within the follicle?
A. Decreased MIT formation
B. Decreased DIT formation
C. Reduced coupling reactions
D. Increased tyrosine iodination

back 47

D. Increased tyrosine iodination

front 48

Chronic TSH excess most strongly causes which cellular adaptation?
A. Reduced follicular cell secretion
B. Increased cell size and secretion
C. Decreased iodide trapping
D. Follicular cell apoptosis

back 48

B. Increased cell size and secretion

front 49

Long-standing TSH stimulation most likely produces which histologic change?
A. Cuboidal to columnar shift
B. Flattened epithelium, scant colloid
C. Loss of follicular architecture
D. C-cell hyperplasia predominates

back 49

A. Cuboidal to columnar shift

front 50

Anterior pituitary TSH secretion is primarily controlled by which hypothalamic hormone?
A. Somatostatin
B. Dopamine
C. Thyrotropin-releasing hormone
D. Corticotropin-releasing hormone

back 50

C. Thyrotropin-releasing hormone

front 51

TRH is best described chemically as:
A. Glycyl-lysyl-arginine-amide
B. Histidyl-prolyl-glycine-amide
C. Leucyl-tyrosyl-alanine-amide
D. Pyroglutamyl-histidylproline-amide

back 51

D. Pyroglutamyl-histidylproline-amide

front 52

TSH receptor activation primarily signals through:
A. IP3/DAG pathway
B. cAMP second messenger
C. JAK/STAT signaling
D. Tyrosine kinase signaling

back 52

B. cAMP second messenger

front 53

TRH receptor activation primarily uses:
A. cGMP second messenger
B. cAMP second messenger
C. Phospholipase C pathway
D. Nuclear receptor binding

back 53

C. Phospholipase C pathway

front 54

A child exposed to cold has increased thyroid drive. Best-known trigger upstream of TSH is:
A. Increased cortisol secretion
B. Increased dopamine secretion
C. Increased somatostatin release
D. Increased TRH secretion

back 54

D. Increased TRH secretion

front 55

Which set lists the best-known antithyroid drugs?
A. Methimazole, levothyroxine, iodine
B. Propranolol, lithium, amiodarone
C. Thiocyanate, PTU, iodides
D. T3, T4, calcitonin

back 55

C. Thiocyanate, PTU, iodides

front 56

Thiocyanate ions primarily decrease which thyroid process?
A. Coupling of iodotyrosines
B. Iodide trapping
C. Proteolysis of thyroglobulin
D. Peripheral T4 to T3 conversion

back 56

B. Iodide trapping

front 57

Chronic blockade of thyroid secretion by thiocyanate can lead to:
A. Myxedema coma
B. Thyroid storm
C. Exophthalmos
D. Goiter

back 57

D. Goiter

front 58

Which drug prevents thyroid hormone formation from iodides and tyrosine?
A. Propylthiouracil
B. Levothyroxine
C. Calcitonin
D. Liothyronine

back 58

A. Propylthiouracil

front 59

Iodides in very high concentration most consistently:
A. Increase iodide trapping
B. Increase TSH secretion
C. Decrease all thyroid activity phases
D. Increase coupling reactions

back 59

C. Decrease all thyroid activity phases

front 60

A patient is scheduled for thyroidectomy in 3 weeks. Why give iodide 2–3 weeks pre-op?
A. Induce autoimmune remission
B. Decrease necessary surgery amount
C. Increase T3 potency at receptors
D. Stimulate follicular hyperplasia

back 60

B. Decrease necessary surgery amount

front 61

The most common form of hyperthyroidism is:
A. Toxic multinodular goiter
B. Subacute thyroiditis
C. Thyroid adenoma
D. Graves’ disease

back 61

D. Graves’ disease

front 62

Graves’ disease is best explained by:
A. TSIs activate TSH receptor cAMP
B. Anti-thyroglobulin blocks hormone release
C. Anti-peroxidase increases iodide oxidation
D. TSH receptor mutations reduce signaling

back 62

A. TSIs activate TSH receptor cAMP

front 63

Hyperthyroidism arising within thyroid tissue that secretes large hormone quantities is:
A. Thyroid adenoma
B. Hashimoto disease
C. Endemic goiter
D. Subacute thyroiditis

back 63

A. Thyroid adenoma

front 64

A patient with excess thyroid hormone most likely reports:
A. Cold intolerance
B. High excitability
C. Bradykinesia
D. Hypersomnia

back 64

B. High excitability

front 65

A patient with thyrotoxicosis most characteristically develops:
A. Heat intolerance
B. Heat-seeking behavior
C. Decreased skin perfusion
D. Reduced sweat production

back 65

A. Heat intolerance

front 66

A hyperthyroid patient develops frequent loose stools. This symptom is best described as:
A. Constipation predominant
B. Steatorrhea predominant
C. Hematochezia predominant
D. Varying degrees of diarrhea

back 66

D. Varying degrees of diarrhea

front 67

Which finding best reflects hyperthyroid skeletal muscle effects?
A. Hyperreflexia
B. Muscle weakness
C. Increased muscle bulk
D. Spastic rigidity

back 67

B. Muscle weakness

front 68

A patient is “exhausted but can’t sleep.” This pattern best fits:
A. Hypothyroid hypersomnia
B. Major depression hypersomnia
C. Fatigue with insomnia
D. Obstructive sleep apnea

back 68

C. Fatigue with insomnia

front 69

A hyperthyroid patient’s hands shake when holding a cup. This is:
A. Tremor of hands
B. Asterixis of hands
C. Clonus of wrists
D. Chorea of fingers

back 69

A. Tremor of hands

front 70

“Protrusion of eyeballs” in hyperthyroidism is called:
A. Ptosis
B. Enophthalmos
C. Nystagmus
D. Exophthalmos

back 70

D. Exophthalmos

front 71

Exophthalmos accompanied by circulating immunoglobulins suggests:
A. Toxic adenoma mechanism
B. Autoimmune process
C. Iodine deficiency mechanism
D. Drug-induced thyroiditis

back 71

B. Autoimmune process

front 72

The most accurate diagnostic test for hyperthyroidism listed here is:
A. Basal metabolic rate
B. Serum TSH by RIA
C. Free thyroxine level
D. Serum TSI by RIA

back 72

C. Free thyroxine level

front 73

Which test is specifically noted as measured by radioimmunoassay?
A. Serum TSH concentration
B. Basal metabolic rate
C. Stool fat quantification
D. Reflex tendon latency

back 73

A. Serum TSH concentration

front 74

In suspected Graves disease, which antibody assay is noted as measured by radioimmunoassay?
A. Anti-centromere
B. Anti-dsDNA
C. Anti-mitochondrial
D. Thyroid-stimulating immunoglobulin

back 74

D. Thyroid-stimulating immunoglobulin

front 75

The most direct treatment for hyperthyroidism is:
A. Propylthiouracil therapy
B. Surgical thyroid removal
C. Cold exposure avoidance
D. Dietary iodine supplementation

back 75

B. Surgical thyroid removal

front 76

A hyperplastic thyroid is treated most directly with:
A. Oral calcium carbonate
B. Levothyroxine replacement
C. Radioactive iodide therapy
D. Dopamine agonist therapy

back 76

C. Radioactive iodide therapy

front 77

The autoimmune cause of hypothyroidism is:
A. Hashimoto’s disease
B. Graves’ disease
C. Thyroid adenoma
D. Endemic goiter

back 77

A. Hashimoto’s disease

front 78

Progressive deterioration and fibrosis of the thyroid gland is most consistent with:
A. Adenoma degeneration
B. Thyroid hormone resistance
C. Iodide excess toxicity
D. Thyroiditis

back 78

D. Thyroiditis

front 79

In regions with insufficient iodine intake, people commonly develop:
A. Graves disease
B. Thyroid adenoma
C. Endemic goiters
D. Hashimoto disease

back 79

C. Endemic goiters

front 80

A colloid goiter can result from which abnormality?
A. Deficient iodide-trapping mechanism
B. Excess calcitonin secretion
C. Excess TRH secretion
D. Increased T3 receptor affinity

back 80

A. Deficient iodide-trapping mechanism

front 81

A colloid goiter can result from which abnormality?
A. Excess iodide organification
B. Deficient peroxidase system
C. Excess coupling of iodotyrosines
D. Increased deiodinase recycling

back 81

B. Deficient peroxidase system

front 82

A colloid goiter can result from which abnormality?
A. Increased iodide trapping
B. Increased thyroglobulin proteolysis
C. Increased TSH receptor cAMP
D. Deficient coupling of iodotyrosines

back 82

D. Deficient coupling of iodotyrosines

front 83

A colloid goiter can result from which abnormality?
A. Excess pendrin transport
B. Excess thyroid peroxidase
C. Deiodinase enzyme deficiency
D. Excess plasma protein binding

back 83

C. Deiodinase enzyme deficiency

front 84

Some foods contain substances with PTU-like activity causing TSH-stimulated thyroid enlargement. These are:
A. Thyroid-stimulating immunoglobulins
B. Goitrogenic substances
C. Thyroxine-binding globulins
D. Inorganic iodides

back 84

B. Goitrogenic substances

front 85

An adult has near-total thyroid hormone loss with swelling and a baggy face. This condition is:
A. Graves disease
B. Myxedema
C. Thyroid adenoma
D. Subacute thyroiditis

back 85

B. Myxedema

front 86

The edema in myxedema is best explained by excess interstitial:
A. Albumin leakage
B. Sodium chloride crystals
C. Lipid droplets
D. Hyaluronic–chondroitin protein gel

back 86

D. Hyaluronic–chondroitin protein gel

front 87

Which hypothyroid test pattern best matches classic teaching?
A. Low free T4, high TSH
B. High free T4, low TSH
C. Low TSH, high BMR
D. High TSH, high BMR

back 87

A. Low free T4, high TSH

front 88

Proper long-term treatment for primary hypothyroidism is:
A. Calcitonin replacement
B. TRH supplementation
C. Thyroxine administration
D. Propylthiouracil therapy

back 88

C. Thyroxine administration

front 89

Extreme hypothyroidism during childhood causing poor growth and mental retardation is:
A. Myxedema
B. Graves disease
C. Thyroid storm
D. Cretinism

back 89

D. Cretinism

front 90

The two major categories of cretinism are:
A. Autoimmune and iatrogenic
B. Congenital and endemic
C. Toxic and nontoxic
D. Central and peripheral

back 90

B. Congenital and endemic

front 91

A neonate with cretinism must receive which therapy early to prevent deficits?
A. Iodine or thyroxine early
B. Radioiodine ablation early
C. Propylthiouracil early
D. Surgery within first weeks

back 91

A. Iodine or thyroxine early

front 92

In cretinism, which growth pattern best explains “short, stocky, obese” appearance?
A. Soft tissue more inhibited
B. Both equally inhibited
C. Skeletal more inhibited
D. Skeletal unaffected, soft tissue

back 92

C. Skeletal more inhibited

front 93

Which pairing best matches thyroid outputs and functions?
A. T3 lowers calcium, T4 raises calcium
B. Calcitonin raises calcium, T4 lowers calcium
C. T3 lowers calcium, calcitonin raises rate
D. T3/T4 raise rate, calcitonin lowers calcium

back 93

D. T3/T4 raise rate, calcitonin lowers calcium

front 94

Sodium-iodide symporter is located on the:
A. Basolateral plasma membrane
B. Apical follicular membrane
C. Nuclear membrane
D. Mitochondrial inner membrane

back 94

A. Basolateral plasma membrane

front 95

Energy driving iodide uptake via NIS comes from:
A. Direct ATP hydrolysis by NIS
B. Chloride gradient across apical side
C. Na-K ATPase sodium gradient
D. Iodine oxidation releasing energy

back 95

C. Na-K ATPase sodium gradient

front 96

As thyroid activity increases, intracellular iodide becomes:
A. Lower than blood
B. Similar to blood
C. Slightly below blood
D. Higher than blood

back 96

D. Higher than blood

front 97

Which hormone increases NIS activity?
A. Calcitonin
B. Thyroid-stimulating hormone
C. Somatostatin
D. Prolactin

back 97

B. Thyroid-stimulating hormone

front 98

Thyroid epithelial cells secrete into follicle a protein providing tyrosines for iodination:
A. Albumin
B. Calcitonin
C. Thyroglobulin
D. Thyroxine-binding prealbumin

back 98

C. Thyroglobulin

front 99

Two organelles key for thyroglobulin synthesis/secretion are:
A. Smooth ER and lysosome
B. Rough ER and Golgi
C. Nucleus and mitochondria
D. Ribosome and peroxisome

back 99

B. Rough ER and Golgi

front 100

In presence of hydrogen peroxide, which enzyme converts iodide to iodine?
A. Deiodinase
B. Pendrin
C. Megalin
D. Peroxidase

back 100

D. Peroxidase

front 101

After coupling, which hormone remains part of the thyroglobulin molecule?
A. Thyroxine (T4)
B. Triiodothyronine (T3)
C. Calcitonin
D. Reverse T3

back 101

A. Thyroxine (T4)

front 102

The storage form of thyroid hormone not released into blood is:
A. Free T3
B. Free T4
C. Thyroglobulin
D. Thyroxine-binding globulin

back 102

C. Thyroglobulin

front 103

Release of T3/T4 from thyroglobulin begins with:
A. Exocytosis of secretory granules
B. Pinocytosis of colloid
C. Diffusion across apical membrane
D. Transcriptional activation

back 103

B. Pinocytosis of colloid

front 104

The enzymatic step liberating free T3/T4 uses:
A. Phosphatases
B. Ligases
C. Deiodinases
D. Lysosomal proteases

back 104

D. Lysosomal proteases

front 105

Luminal membrane protein mediating thyroglobulin endocytosis/transcytosis is:
A. Megalin
B. Pendrin
C. NIS
D. TBG

back 105

A. Megalin

front 106

The specific glycosaminoglycans increased in myxedema include:
A. Keratan sulfate and dermatan sulfate
B. Heparin and heparan sulfate
C. Hyaluronidase and collagenase
D. Hyaluronic acid and chondroitin sulfate

back 106

D. Hyaluronic acid and chondroitin sulfate

front 107

In primary hypothyroidism, which lab change is expected?
A. Decreased TSH secretion
B. Increased TSH secretion
C. Increased free T4
D. Increased basal metabolic rate

back 107

B. Increased TSH secretion

front 108

The “baggy face” of severe hypothyroidism is most directly due to:
A. Acute capillary hemorrhage
B. Lipid deposition in dermis
C. Interstitial glycosaminoglycan gel
D. Excess adrenergic vasodilation

back 108

C. Interstitial glycosaminoglycan gel

front 109

Which thyroid hormone binds plasma proteins most avidly?
A. T3
B. Calcitonin
C. T4
D. Thyroglobulin

back 109

C. T4

front 110

Higher plasma-binding affinity best matches which release pattern?
A. T4 1 day; T3 6 days
B. T4 3 days; T3 3 days
C. T4 1 day; T3 1 day
D. T4 6 days; T3 1 day

back 110

D. T4 6 days; T3 1 day

front 111

A thyroid preparation is slower acting with longer latent period and longer half-life. Which hormone is it?
A. T4
B. T3
C. Calcitonin
D. Thyroglobulin

back 111

A. T4

front 112

Thyroid hormone increases functional activity throughout the body primarily by:
A. Opening ligand-gated channels
B. Activating membrane GPCRs
C. Increasing gene transcription
D. Inhibiting DNA replication

back 112

C. Increasing gene transcription

front 113

The thyroid hormone that directly binds intracellular thyroid receptors to drive transcription is:
A. T4
B. T3
C. Calcitonin
D. Thyroglobulin

back 113

B. T3

front 114

Over 90% of thyroid hormone bound to receptors is:
A. T3
B. T4
C. MIT
D. DIT

back 114

A. T3

front 115

The TR/RXR complex initiates transcription by binding:
A. TATA boxes
B. TREs
C. EREs
D. GREs

back 115

B. TREs

front 116

Increased “leakiness” of which ion most raises heat production and BMR?
A. Na+
B. K+
C. Cl−
D. Ca2+

back 116

A. Na+

front 117

A child becomes tall early; bones mature rapidly; epiphyses close early; adult height may be reduced. Most consistent thyroid state:
A. Hypothyroidism
B. Euthyroid
C. Thyroid hormone resistance
D. Hyperthyroidism

back 117

D. Hyperthyroidism

front 118

Thyroid hormone effect on carbohydrate and fat metabolism is:
A. Decreases both pathways
B. Increases CHO only
C. Increases both pathways
D. Increases fat only

back 118

C. Increases both pathways

front 119

In hypothyroidism, expected lipid pattern is:
A. ↑chol, ↑PL, ↑TG; ↓FFA
B. ↓chol, ↓PL, ↓TG; ↑FFA
C. ↑FFA, ↓chol, ↓TG
D. ↓PL, ↓chol; ↑FFA

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A. ↑chol, ↑PL, ↑TG; ↓FFA

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Thyroid hormone lowers cholesterol partly by:
A. Decreasing bile cholesterol secretion
B. ↑bile secretion, ↑fecal loss
C. Increasing intestinal cholesterol absorption
D. Blocking hepatic bile formation

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B. ↑bile secretion, ↑fecal loss

front 121

Thyroid hormone lowers LDL largely by increasing hepatic:
A. Lipoprotein lipase activity
B. VLDL secretion rate
C. ApoA-I synthesis
D. LDL receptor expression

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D. LDL receptor expression

front 122

Normal thyroid hormone effect on free fatty acids is:
A. Increases free fatty acids
B. Decreases free fatty acids
C. No change in free acids
D. Eliminates plasma free acids

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A. Increases free fatty acids

front 123

Excess thyroid hormone raises vitamin requirements mainly because it:
A. Suppresses enzyme production
B. Decreases transcriptional activity
C. Increases enzyme production
D. Blocks vitamin hepatic storage

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C. Increases enzyme production

front 124

Which hyperthyroid pattern is most expected?
A. ↓BMR, ↓weight
B. ↑BMR, ↓weight
C. ↓BMR, ↑weight
D. ↑BMR, ↑weight

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B. ↑BMR, ↓weight

front 125

In hyperthyroidism, blood flow and cardiac output typically:
A. Decrease both
B. Increase flow, decrease output
C. Decrease flow, increase output
D. Increase both

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D. Increase both

front 126

In hyperthyroidism, heart rate and contractile strength:
A. Increase both
B. Decrease both
C. Increase rate, decrease strength
D. Decrease rate, increase strength

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A. Increase both

front 127

Thyroid hormone has little net effect on mean arterial pressure because:
A. Both systolic and diastolic rise
B. Both systolic and diastolic fall
C. Systolic rises, diastolic falls
D. Diastolic rises, systolic falls

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C. Systolic rises, diastolic falls

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Which combination best maintains mean arterial pressure with thyroid hormone?
A. ↓systolic, ↑diastolic; ↓pulse pressure
B. ↑systolic, ↑diastolic; ↓pulse pressure
C. ↓systolic, ↓diastolic; ↑pulse pressure
D. ↑systolic, ↓diastolic; ↑pulse pressure

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D. ↑systolic, ↓diastolic; ↑pulse pressure

front 129

Excess thyroid hormone effect on respiration is:
A. Decreases rate and depth
B. Increases rate and depth
C. Increases depth only
D. Increases rate only

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B. Increases rate and depth

front 130

Excess thyroid hormone effect on GI motility is:
A. Increased motility
B. Decreased motility
C. No change in motility
D. Complete motility paralysis

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A. Increased motility

front 131

Hyperthyroidism effect on CNS most resembles:
A. Psychomotor slowing, apathy
B. CNS depression, confusion
C. Flaccid paralysis, coma
D. CNS stimulation, anxiety

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D. CNS stimulation, anxiety

front 132

Hyperthyroidism effect on muscle mass is:
A. Increased protein synthesis
B. Increased muscle hypertrophy
C. Decreased via catabolism
D. Unchanged muscle protein turnover

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C. Decreased via catabolism

front 133

A patient with chronic diarrhea is more consistent with:
A. Hypothyroidism
B. Hyperthyroidism
C. Isolated calcitonin excess
D. Addison disease

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B. Hyperthyroidism

front 134

A patient with constipation is more consistent with:
A. Hyperthyroidism
B. Euthyroid state
C. Graves disease
D. Hypothyroidism

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D. Hypothyroidism

front 135

Hyperthyroid tremor is best described as:
A. Fine “paper vibration” tremor
B. Coarse “shivering” tremor
C. Intention tremor
D. Resting pill-rolling tremor

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A. Fine “paper vibration” tremor

front 136

Parkinson tremor is best described as:
A. Fine paper vibration
B. Pure intention tremor
C. Coarse shivering tremor
D. Only postural tremor

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C. Coarse shivering tremor

front 137

A patient sleeping 12–14 hours daily most suggests:
A. Hypothyroidism
B. Hyperthyroidism
C. Thyroid storm
D. Anxiety disorder

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A. Hypothyroidism

front 138

del

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del

front 139

In men, lack vs excess thyroid hormone most classically causes:
A. Lack impotence; excess libido
B. Lack libido gain; excess loss
C. Lack oliguria; excess polyuria
D. Lack libido loss; excess impotence

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D. Lack libido loss; excess impotence

front 140

In women, thyroid hormone deficiency is associated with:
A. Oligomenorrhea
B. Amenorrhea
C. Menorrhagia, polymenorrhea, irregular
D. No menstrual changes

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C. Menorrhagia, polymenorrhea, irregular

front 141

In women, excess thyroid hormone is associated with:
A. Menorrhagia and polymenorrhea
B. Oligomenorrhea and amenorrhea
C. Continuous heavy bleeding
D. Regular cycles

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B. Oligomenorrhea and amenorrhea

front 142

TSH stimulation increases which thyroid process?
A. Iodide pump activity
B. Decreased iodide trapping
C. Reduced iodination rate
D. Reduced follicular transport

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A. Iodide pump activity

front 143

TSH stimulation increases which release-related step?
A. Reduced lysosomal activity
B. Decreased colloid uptake
C. Increased hormone deiodination
D. Increased thyroglobulin proteolysis

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D. Increased thyroglobulin proteolysis

front 144

Chronic TSH stimulation increases:
A. Follicular flattening, less secretion
B. Thyroid cell number increases
C. C-cell number increases
D. Follicles disappear completely

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B. Thyroid cell number increases

front 145

TSH increases hormone synthesis partly by:
A. Increasing tyrosine iodination
B. Blocking iodide oxidation
C. Decreasing coupling reactions
D. Preventing MIT/DIT formation

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A. Increasing tyrosine iodination

front 146

TSH activates thyroid follicular cells primarily via:
A. JAK-STAT signaling
B. PLC → Ca2+ + DAG
C. cAMP second messenger
D. Tyrosine kinase cascade

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C. cAMP second messenger

front 147

TRH enters hypophysial portal blood from the:
A. Median eminence
B. Supraoptic nucleus
C. Posterior pituitary
D. Anterior pituitary

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A. Median eminence

front 148

TRH triggers TSH release using:
A. cGMP signaling
B. PLC → Ca2+ + DAG
C. cAMP signaling
D. Nuclear receptor binding

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B. PLC → Ca2+ + DAG

front 149

If the hypophysial stalk is cut, cold tolerance is preserved because:
A. TSH is autonomous
B. Thyroid senses cold directly
C. Hypothalamus increases TRH
D. Pituitary stores TRH long-term

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C. Hypothalamus increases TRH

front 150

Which antithyroid drug decreases iodide trapping?
A. Thiocyanate
B. Propylthiouracil
C. High-dose iodides
D. Radioactive iodide

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A. Thiocyanate

front 151

Which antithyroid drug blocks peroxidase and coupling?
A. Thiocyanate
B. Propylthiouracil
C. High-dose iodides
D. Levothyroxine

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B. Propylthiouracil

front 152

Which therapy decreases thyroid activity and size?
A. Thiocyanate
B. Propylthiouracil
C. TRH infusion
D. High-dose iodides

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D. High-dose iodides

front 153

Autoimmune hyperthyroidism with TSIs against TSH receptors is:
A. Hashimoto disease
B. Graves disease
C. Subacute thyroiditis
D. Colloid goiter

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B. Graves disease

front 154

A tumor secretes thyroid hormone, suppressing pituitary TSH. This is:
A. Thyroid adenoma
B. Graves disease
C. Hashimoto disease
D. Endemic goiter

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A. Thyroid adenoma

front 155

Exophthalmos can endanger vision by compressing the:
A. Oculomotor nerve
B. Trochlear nerve
C. Abducens nerve
D. Optic nerve

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D. Optic nerve

front 156

Exophthalmos in thyrotoxicosis is caused mainly by:
A. Corneal edema
B. Lens swelling
C. Retro-orbital edema + myopathy
D. Increased aqueous humor

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C. Retro-orbital edema + myopathy

front 157

In classic thyrotoxicosis, plasma TSH is typically:
A. Absent or very low
B. High with normal T4
C. Normal with high TRH
D. High with high T3

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A. Absent or very low

front 158

Autoimmune destruction causing thyroiditis and goiter is:
A. Graves disease
B. Hashimoto disease
C. Thyroid adenoma
D. Myxedema coma

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B. Hashimoto disease

front 159

Iodine deficiency causes goiter primarily through:
A. Increased calcitonin secretion
B. Increased T3 potency
C. Low T3/T4 → high TSH
D. High TRH → low TSH

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C. Low T3/T4 → high TSH

front 160

Goiter in people without iodine deficiency is:
A. Endemic goiter
B. Graves goiter
C. Toxic multinodular goiter
D. Idiopathic nontoxic colloid goiter

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D. Idiopathic nontoxic colloid goiter

front 161

Which food contains goitrogenic substances?
A. Cabbages or turnips
B. Apples or bananas
C. Beef or chicken
D. Rice or wheat

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A. Cabbages or turnips

front 162

Froglike husky voice with diffuse edema suggests:
A. Graves disease
B. Myxedema
C. Thyroid adenoma
D. Exophthalmos syndrome

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B. Myxedema

front 163

Endemic cretinism most commonly results from:
A. TSH receptor mutation
B. TSIs against TSH receptor
C. Dietary iodine deficiency
D. Pituitary TSH-secreting tumor

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C. Dietary iodine deficiency