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immunology lecture 23 key points

front 1

what is banign?

back 1

unable to invade healthy surrounding tissue, incapable of indefinite growth

front 2

what is malignant?

back 2

becomes progressively more invasive, may metastasize

front 3

what is metastasis?

back 3

the spread of cancer cells beyond the site of origin

front 4

what are the hallmarks of cancer?

back 4

stimulating their own growth

ignoring growth-inhibiting signals

avoiding death by apoptosis

developing a blood supply: angiogenesis

seeding from site of origin to invade other tissues: metastasis

constant replication to expand the tumor-cell population

evasion and outrunning the immune response

front 5

what are the cancers of the immune system/blood?

back 5

leukemia

lymphoma

myeloma

front 6

leukemia

back 6

a group of cancers that begin in the bone marrow and lead to overproduction of white blood cells

front 7

lymphoma

back 7

neoplasms of the lymphatic tissues; usually lymphocytes

front 8

myeloma

back 8

cancer of the plasma cells; they can accumulate in the bone marrow and then secrete abnormal antibodies

front 9

what are proto-oncogenes

back 9

genes that regulate control of cell growth and division; when mutated can contribute to malignant growth of cells

front 10

what are tumor-suppressor genes?

back 10

genes that encode proteins which normally function to prevent cells from becoming cancerous; may also include genes that control apoptosis

front 11

what are the four stages of malignant transformation?

back 11

initiation

promotion

progression

metastasis

front 12

what are the four types of environmental factors that can cause cancer?

back 12

chemicals

UV light

radiation

oncogenic viruses

front 13

what are tumor antigens?

back 13

cells are self cells that may express either unique or inappropriately expressed Ag that can be detected by the immune system

front 14

what are the four groups of tumor antigens that are recognized by the immune system?

back 14

Ag encoded by genes exclusively expressed by tumors

Ag encoded by variant forms of normal genes altered by mutation

Ag normally expressed only at certain stages of development

Ag overexposed in particular tumors

front 15

what are tumor-specific antigens?

back 15

antigens specific to tumor cells, not found in non-cancer cells

front 16

what are tumor-associated antigens?

back 16

normal cellular proteins; not unique to cells

front 17

what are the three phases of immunoediting?

back 17

elimination

equilibrium

escape

front 18

elimination

back 18

also known as immunosurvelliance; innate and adaptive immunity

front 19

equilibrium

back 19

at this stage, the tumor is considered to be dormant; it is decreased in terms of its immunogenicity. the immune system has a hard time recognizing the altered tumor

front 20

escape

back 20

tumor cells with low immunogenicity can evade immune system detection

front 21

how do tumor cells evade immune defenses?

back 21

subversion of MHC class I

sabotage of apoptotic signals

poor co-stimulation

T cell exhaustion of tolerance

front 22

what are the five types of immunity?

back 22

monoclonal antibodies

adaptively transferred DCs

adaptively transferred T cells

CAR T cells

checkpoint/blockade therapy

front 23

monoclonal antibodies

back 23

treatment of terminal B-cell lymphoma with anti-idiotype mAb

mAb against CD20 B-cell marker for non-hodgkin's lymphoma

immunotoxins, toxic molecules coupled to mAb

mAb against HER2 receptor in breast cancer

front 24

adaptively transferred DCs

back 24

isolate autologous DCs

co-culture DCs with antigen

re-infuse antigen loaded DCs into patient

patient has increased CD4+ and CD8+ T cell response against tumor antigens, stimulating a stronger CTL response

front 25

adaptively transferred T cells

back 25

cells obtained from tumors, tumor-draining nodes, or peripheral blood

once obtained, the cells are expanded ex vivo using IL-2 cytokine to overcome anergic states

patients are depleted of lymphocytes to create a niche for the new cells

the expanded cells are reinfused

front 26

what are CAR T cells?

back 26

chimeric antigen receptor

front 27

what are the types of CAR T cells?

back 27

truck

universal CAR

self-driving CAR

armored CAR
self-destruct CAR

conditional CAR

front 28

checkpoint/blockade therapy?

back 28

manipulation of costimulatory signals can improve cancer immunity